Impaired Binding of 14-3-3 to C-Raf in Noonan Syndrome Suggests New Approaches 
in Diseases with Increased Ras Signaling

Molzan, Manuela; Schumacher, Benjamin; Ottmann, Corinna; Baljuls, Angela; Polzien, Lisa; Weyand, Michael; Thiel, Philipp; Rose, Rolf; Rose, Micheline; Kuhenne, Philipp; Kaiser, Markus; Rapp, Ulf R.; Kuhlmann, Jürgen; Ottmann, Christian

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Impaired Binding of 14-3-3 to C-Raf in Noonan Syndrome Suggests New Approaches in Diseases with Increased Ras Signaling

MPS-Authors

Molzan, Manuela
Max Planck Institute of Molecular Physiology, Max Planck Society;

Schumacher, Benjamin
Max Planck Institute of Molecular Physiology, Max Planck Society;

Ottmann, Corinna
Max Planck Institute of Molecular Physiology, Max Planck Society;

Weyand, Michael
Max Planck Institute of Molecular Physiology, Max Planck Society;

Thiel, Philipp
Max Planck Institute of Molecular Physiology, Max Planck Society;

Rose, Rolf
Max Planck Institute of Molecular Physiology, Max Planck Society;

Rose, Micheline
Max Planck Institute of Molecular Physiology, Max Planck Society;

Kuhenne, Philipp
Max Planck Institute of Molecular Physiology, Max Planck Society;

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Kaiser, Markus
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Kuhlmann, Jürgen
Sonstige Wissenschaftliche Organisationseinheiten, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Ottmann, Christian
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Citation

Molzan, M., Schumacher, B., Ottmann, C., Baljuls, A., Polzien, L., Weyand, M., et al. (2010). Impaired Binding of 14-3-3 to C-Raf in Noonan Syndrome Suggests New Approaches in Diseases with Increased Ras Signaling. Molecular and Cellular Biology, 30(19): 1, pp. 4698-4711. Retrieved from http://dx.doi.org/10.1128/MCB.01636-09.

Cite as: https://hdl.handle.net/11858/00-001M-0000-0014-0140-4

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