Decreased repolarization reserve increases defibrillation threshold by favoring early afterdepolarizations in an in silico model of human ventricular tissue
- Author
- Nele Vandersickel (UGent) , Ivan Kazbanov (UGent) , Arne Defauw (UGent) , Daniel A Pijnappels and Alexander Panfilov (UGent)
- Organization
- Keywords
- TORSADES-DE-POINTES, Defibrillation threshold, LONG-QT-SYNDROME, CARDIAC TISSUE, SECONDARY SOURCES, HUMAN HEART, MECHANISM, ARRHYTHMIAS, STIMULATION, BIDOMAIN, MYOCYTES, In alien, Early afterdepolarization, Ventricular fibrillation
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Citation
Please use this url to cite or link to this publication: http://hdl.handle.net/1854/LU-7198868
- MLA
- Vandersickel, Nele, et al. “Decreased Repolarization Reserve Increases Defibrillation Threshold by Favoring Early Afterdepolarizations in an in Silico Model of Human Ventricular Tissue.” HEART RHYTHM, vol. 12, no. 5, 2015, pp. 1088–96, doi:10.1016/j.hrthm.2015.01.033.
- APA
- Vandersickel, N., Kazbanov, I., Defauw, A., Pijnappels, D. A., & Panfilov, A. (2015). Decreased repolarization reserve increases defibrillation threshold by favoring early afterdepolarizations in an in silico model of human ventricular tissue. HEART RHYTHM, 12(5), 1088–1096. https://doi.org/10.1016/j.hrthm.2015.01.033
- Chicago author-date
- Vandersickel, Nele, Ivan Kazbanov, Arne Defauw, Daniel A Pijnappels, and Alexander Panfilov. 2015. “Decreased Repolarization Reserve Increases Defibrillation Threshold by Favoring Early Afterdepolarizations in an in Silico Model of Human Ventricular Tissue.” HEART RHYTHM 12 (5): 1088–96. https://doi.org/10.1016/j.hrthm.2015.01.033.
- Chicago author-date (all authors)
- Vandersickel, Nele, Ivan Kazbanov, Arne Defauw, Daniel A Pijnappels, and Alexander Panfilov. 2015. “Decreased Repolarization Reserve Increases Defibrillation Threshold by Favoring Early Afterdepolarizations in an in Silico Model of Human Ventricular Tissue.” HEART RHYTHM 12 (5): 1088–1096. doi:10.1016/j.hrthm.2015.01.033.
- Vancouver
- 1.Vandersickel N, Kazbanov I, Defauw A, Pijnappels DA, Panfilov A. Decreased repolarization reserve increases defibrillation threshold by favoring early afterdepolarizations in an in silico model of human ventricular tissue. HEART RHYTHM. 2015;12(5):1088–96.
- IEEE
- [1]N. Vandersickel, I. Kazbanov, A. Defauw, D. A. Pijnappels, and A. Panfilov, “Decreased repolarization reserve increases defibrillation threshold by favoring early afterdepolarizations in an in silico model of human ventricular tissue,” HEART RHYTHM, vol. 12, no. 5, pp. 1088–1096, 2015.
@article{7198868,
author = {{Vandersickel, Nele and Kazbanov, Ivan and Defauw, Arne and Pijnappels, Daniel A and Panfilov, Alexander}},
issn = {{1547-5271}},
journal = {{HEART RHYTHM}},
keywords = {{TORSADES-DE-POINTES,Defibrillation threshold,LONG-QT-SYNDROME,CARDIAC TISSUE,SECONDARY SOURCES,HUMAN HEART,MECHANISM,ARRHYTHMIAS,STIMULATION,BIDOMAIN,MYOCYTES,In alien,Early afterdepolarization,Ventricular fibrillation}},
language = {{eng}},
number = {{5}},
pages = {{1088--1096}},
title = {{Decreased repolarization reserve increases defibrillation threshold by favoring early afterdepolarizations in an in silico model of human ventricular tissue}},
url = {{http://doi.org/10.1016/j.hrthm.2015.01.033}},
volume = {{12}},
year = {{2015}},
}
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