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Mutation in the transcriptional regulator PhoP contributes to avirulence of Mycobacterium tuberculosis H37Ra strain

MPS-Authors
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Lee,  Jong Seok
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Krause,  Roland
Department of Cellular Microbiology, Max Planck Institute for Infection Biology;
Dept. of Computational Molecular Biology (Head: Martin Vingron), Max Planck Institute for Molecular Genetics, Max Planck Society;

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Schreiber,  Jörg
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Mollenkopf,  Hans J.
Core Facilities / Microarray, Max Planck Institute for Infection Biology, Max Planck Society;

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Kowall,  Jane
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Stein,  Robert
Core Facilities / Proteinanalysis, Max Planck Institute for Infection Biology, Max Planck Society;

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Patron,  Juan Pablo
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Jorg,  Sabine
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaufmann,  Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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引用

Lee, J. S., Krause, R., Schreiber, J., Mollenkopf, H. J., Kowall, J., Stein, R., Jeon, B.-Y., Kwak, J.-Y., Song, M.-K., Patron, J. P., Jorg, S., Roh, K., Cho, S.-N., & Kaufmann, S. H. E. (2008). Mutation in the transcriptional regulator PhoP contributes to avirulence of Mycobacterium tuberculosis H37Ra strain. Cell Host & Microbe, 3(2), 97-103.


引用: https://hdl.handle.net/11858/00-001M-0000-000E-C1F3-7
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