Double heterozygous mutation of KCNH2 and ANK2 leads to phenotypical 
aggravation of congenital long QT syndrome

Schweizer, Patrick; Runge, Sarah; Gessner, Guido; Heinemann, Stefan H.; Zehelein, Joerg; Koenen, Michael; Khalil, Markus; Ulmer, Herbert E.; Koenen, Michael; Katus, Hugo A.; Becker, Rüdiger; Thomas, Dierck

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Double heterozygous mutation of KCNH2 and ANK2 leads to phenotypical aggravation of congenital long QT syndrome

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Schweizer, Patrick
Department of Cell Physiology, Max Planck Institute for Medical Research, Max Planck Society;
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Zehelein, Joerg
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Koenen, Michael
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;
Department of Cell Physiology, Max Planck Institute for Medical Research, Max Planck Society;

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Schweizer, P., Runge, S., Gessner, G., Heinemann, S. H., Zehelein, J., Koenen, M., Khalil, M., Ulmer, H. E., Koenen, M., Katus, H. A., Becker, R., & Thomas, D. (2010). Double heterozygous mutation of KCNH2 and ANK2 leads to phenotypical aggravation of congenital long QT syndrome. Poster presented at Heart Rhythm 2010.

引用: https://hdl.handle.net/11858/00-001M-0000-002E-8018-8

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