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1,8-Cineole promotes G0/G1 cell cycle arrest and oxidative stress-induced senescence in HepG2 cells and sensitizes cells to anti-senescence drugs.
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Document type | Article de périodique (Journal article) – Article de recherche |
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Access type | Accès libre |
Publication date | 2020 |
Language | Anglais |
Journal information | "Life sciences" - Vol. 243, p. 117271 [1-13] (2020) |
Peer reviewed | yes |
Publisher | Elsevier ((Netherlands) Amsterdam) |
issn | 0024-3205 |
e-issn | 1879-0631 |
Publication status | Publié |
Affiliations |
UCL
- SSS/IREC/GAEN - Pôle d'Hépato-gastro-entérologie UCL - (SLuc) Service d'hépato-gastro-entérologie |
MESH Subject | Antioxidants ; Cell Cycle Checkpoints ; Cellular Senescence ; Dose-Response Relationship, Drug ; Enzyme Induction ; Eucalyptol ; G1 Phase ; Hep G2 Cells ; Humans ; Oxidative Stress ; Protein Kinases ; Reactive Oxygen Species ; Resting Phase, Cell Cycle ; Ribosomal Protein S6 Kinases, 70-kDa ; TOR Serine-Threonine Kinases |
Keywords | 1,8-Cineole ; AMPK ; Akt/mTOR ; Cell cycle arrest ; Hepatocellular carcinoma cells ; MAPKs ; Oxidative stress ; Senescence |
Links |
Bibliographic reference | Rodenak-Kladniew, Boris ; Castro, Agustina ; Starkel, Peter ; Galle, Marianela ; Crespo, Rosana. 1,8-Cineole promotes G0/G1 cell cycle arrest and oxidative stress-induced senescence in HepG2 cells and sensitizes cells to anti-senescence drugs.. In: Life sciences, Vol. 243, p. 117271 [1-13] (2020) |
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Permanent URL | http://hdl.handle.net/2078.1/230967 |