The Glucose-Dependent Insulinotropic Polypeptide Receptor is Overexpressed 
Amongst GNAS1 Mutation-Negative Somatotropinomas and Drives Growth Hormone 
(GH)-Promoter Activity in GH3 Cells

Occhi, G.; Losa, M.; Albiger, N.; Trivellin, G.; Regazzo, D.; Scanarini, M.; Monteserin-Garcia, J. L.; Fröhlich, B.; Ferasin, S.; Terreni, M. R.; Fassina, A.; Vitiello, L.; Stalla, G.; Mantero, F.; Scaroni, C.

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The Glucose-Dependent Insulinotropic Polypeptide Receptor is Overexpressed Amongst GNAS1 Mutation-Negative Somatotropinomas and Drives Growth Hormone (GH)-Promoter Activity in GH3 Cells

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Monteserin-Garcia, J. L.
AG Stalla, Günter, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Stalla, G.
AG Stalla, Günter, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Occhi, G., Losa, M., Albiger, N., Trivellin, G., Regazzo, D., Scanarini, M., et al. (2011). The Glucose-Dependent Insulinotropic Polypeptide Receptor is Overexpressed Amongst GNAS1 Mutation-Negative Somatotropinomas and Drives Growth Hormone (GH)-Promoter Activity in GH3 Cells. Journal of Neuroendocrinology, 23(7), 641-649.

Cite as: https://hdl.handle.net/11858/00-001M-0000-000E-8E7C-D

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